| Protein | Protein Name | Molecular Type | Hallmark | Feature | Evidence | Reference |
|---|
| EPHA2 | EPH receptor A2 | Receptor Tyrosine Kinase | Genome Instability, Mutation & Perturbation | Overexpression | EPHA2 is overexpressed in prostate cancer. | Reference |
| EPHA2 | EPH receptor A2 | Receptor Tyrosine Kinase | Metastasis | Metastasis | EPHA2 is involved in prostate cancer metastasis through EphrinA1-EPHA2-VAV3-RAC1 signaling axis. | Reference |
| EPHA2 | EPH receptor A2 | Receptor Tyrosine Kinase | Metastasis | Cell Motility | EPHA2 is involved in prostate cancer cell motility as overexpression of EPHA2 promotes prostate cancer cell motility in PC-3 cells in ligand independent manner. | Reference |
| EPHA2 | EPH receptor A2 | Receptor Tyrosine Kinase | Metastasis | Cell Motility | EPHA2 is involved in prostate cancer cell motility by adopting an amoboid movement based on Rho-mediated cell rounding. | Reference |
| EPHA2 | EPH receptor A2 | Receptor Tyrosine Kinase | Metastasis | Cell Adhesion | EPHA2 is involved in regulation of cell adhesion and cytoskeletal rearrangement in prostate cancer. | Reference |
| EPHA2 | EPH receptor A2 | Receptor Tyrosine Kinase | Metastasis | Cell Migration | Arachidonic acid mediates prostate cancer epithelial cell migration in an EPHA2 dependent fashion. | Reference |
| EPHA2 | EPH receptor A2 | Receptor Tyrosine Kinase | Metastasis | Bone Metastasis | EPHA2 is associated with bone metastasis in prostate cancer. | Reference |
| EPHA2 | EPH receptor A2 | Receptor Tyrosine Kinase | Metastasis | Bone Metastasis | It is involved in prostate cancer bone metastasis through EphA2-Vav3-Rac1 signaling axis. | Reference |