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You've got <font color ="red">14</font> hits!<HR width="580" align="left"> <table border='1' cellpadding="0" cellspacing="0" bordercolor="#000060" width =580>
<tr>
  <td  bgcolor = "#47589D"><font color = "white"><b>Gene 1</b></font></td>
  <td bgcolor = "#47589D"><font color = "white"><b>Gene 2</b></font></td>
  <td bgcolor = "#47589D"><font color = "white"><b>Interaction Type</b></font></td>
 <td bgcolor = "#47589D"><font color = "white"><b>Sentence</b></font></td>
 <td bgcolor = "#47589D"><font color = "white"><b>Value</b></font></td>
 <td bgcolor = "#47589D"><font color = "white"><b>Judge me</b></font></td>
 </tr>
<tr >
  <td>Notch</td>
  <td>decapentaplegic</td>
  <td>interact2</td>
  <td>Genetic combinations between mutations that increase or reduce Notch , veinlet and decapentaplegic activities suggest that the maintenance of the vein differentiation state during pupal development in</td>
  <td>14</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=219">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=219">bad</a>
                </td>
 </tr>
<tr bgcolor=#CCCCCC>
  <td>Notch</td>
  <td>Delta</td>
  <td>interact2</td>
  <td>This function is triggered by a ligand , Delta , and is implemented by the release of the intracellular domain of Notch from the membrane and by its interaction with the protein Suppressor of Hairless</td>
  <td>14</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=2094">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=2094">bad</a>
                </td>
 </tr>
<tr >
  <td>Notch</td>
  <td>Wingless</td>
  <td>associate1</td>
  <td>In particular , in Drosophila , there is evidence that a Su ( H ) -independent activity of Notch is associated with Wingless signaling .</td>
  <td>14</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=2095">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=2095">bad</a>
                </td>
 </tr>
<tr bgcolor=#CCCCCC>
  <td>Notch</td>
  <td>dishevelled</td>
  <td>interact1</td>
  <td>Genetic analysis demonstrates that the dishevelled gene , which encodes a molecule previously implicated in implementation of the Winglass signal , interacts antagonistically with Notch and one of its</td>
  <td>13</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=387">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=387">bad</a>
                </td>
 </tr>
<tr >
  <td>Notch</td>
  <td>Dsh</td>
  <td>bind1</td>
  <td>These novel Notch alleles encode truncated receptors lacking the carboxy terminus of the NICD , which is the binding site for the repressor Dishevelled ( Dsh ) .</td>
  <td>13</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=1346">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=1346">bad</a>
                </td>
 </tr>
<tr bgcolor=#CCCCCC>
  <td>Notch</td>
  <td>DPP</td>
  <td>interact3</td>
  <td>No dominant interaction is observed with sensitized conditions in the Wnt , Notch , Insulin-Pi3K , and DPP signalling pathways .</td>
  <td>12</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=894">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=894">bad</a>
                </td>
 </tr>
<tr >
  <td>Notch</td>
  <td>Ubx</td>
  <td>bind1</td>
  <td>This response to Notch is likely to be mediated by the dTcf binding sites in the Ubx ( VM ) B enhancer .</td>
  <td>12</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=2096">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=2096">bad</a>
                </td>
 </tr>
<tr bgcolor=#CCCCCC>
  <td>Notch</td>
  <td>decapentaplegic</td>
  <td>interact2</td>
  <td>Genetic combinations between mutations that increase or reduce Notch , veinlet and decapentaplegic activities suggest that the maintenance of the vein differentiation state during pupal development in</td>
  <td>11</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=2179">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=2179">bad</a>
                </td>
 </tr>
<tr >
  <td>Notch</td>
  <td>DPP</td>
  <td>interact3</td>
  <td>No dominant interaction is observed with sensitized conditions in the Wnt , Notch , Insulin-Pi3K , and DPP signalling pathways .</td>
  <td>11</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=111">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=111">bad</a>
                </td>
 </tr>
<tr bgcolor=#CCCCCC>
  <td>Notch</td>
  <td>DPP</td>
  <td>interact3</td>
  <td>No dominant interaction is observed with sensitized conditions in the Wnt , Notch , Insulin-Pi3K , and DPP signalling pathways .</td>
  <td>11</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=1714">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=1714">bad</a>
                </td>
 </tr>
<tr >
  <td>Notch</td>
  <td>Ras</td>
  <td>interact3</td>
  <td>Reciprocal regulatory interactions between the Notch and Ras signaling pathways in the Drosophila embryonic mesoderm .</td>
  <td>11</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=1585">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=1585">bad</a>
                </td>
 </tr>
<tr bgcolor=#CCCCCC>
  <td>Notch</td>
  <td>Wingless</td>
  <td>associate1</td>
  <td>In particular , in Drosophila , there is evidence that a Su ( H ) -independent activity of Notch is associated with Wingless signaling .</td>
  <td>11</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=73">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=73">bad</a>
                </td>
 </tr>
<tr >
  <td>Notch</td>
  <td>DPP</td>
  <td>interact3</td>
  <td>No dominant interaction is observed with sensitized conditions in the Wnt , Notch , Insulin-Pi3K , and DPP signalling pathways .</td>
  <td>10</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=2116">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=2116">bad</a>
                </td>
 </tr>
<tr bgcolor=#CCCCCC>
  <td>Notch</td>
  <td>Dishevelled</td>
  <td>bind1</td>
  <td>These novel Notch alleles encode truncated receptors lacking the carboxy terminus of the NICD , which is the binding site for the repressor Dishevelled ( Dsh ) .</td>
  <td>9</td>
  <td><a href ="vote.php?vote=1&textfield=Notch&id=395">good</a>
                <a href="vote.php?vote=-1&textfield=Notch&id=395">bad</a>
                </td>
 </tr>
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    <td width="580" height="77" valign="top"><hr>
      <div align="center"><i><a href="index.html">Text2Knowledge</a> <font color = "red"> ... </font><a href="http://bioinformatics.org/~hyy/index.html">Yong Huang</a> 
        2002</i></div>
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