Role of HOX and MEIS1 genes in induction of acute myeloid leukemia  Thorsteinsdottir U, Kroon E, Jerome L, Blasi F, Sauvageau G.

Hoxa9 evokes a cytokine-selective block in differentiation by a mechanism that does not require Meis gene expression  Calvo KR, Sykes DB, Pasillas M, Kamps MP.

Biochemical interactions between HoxA9 and TALE homeoproteins mediate cellular transformation in hematopoietic cells  Schnabel CA, Jacobs Y, Cleary ML

Upregulation of Meis1 and HoxA9 in acute lymphocytic leukemias with the t(4 : 11) abnormality Rozovskaia T, Feinstein E, Mor O, Foa R, Blechman J, Nakamura T, Croce CM, Cimino G, Canaani E.

Expression of the NUP98/HOXA9 fusion transcript in the blast crisis  Yamamoto K, Nakamura Y, Saito K, Furusawa S.

Chronic myelomonocytic leukemia with t(7;11)(p15;p15) and NUP98/HOXA9 fusion  Wong KF, So CC, Kwong YL.

Translocation (7;11)(p15;p15) is uncommon in acute myeloid leukemia  Kwong YL, Pang A

The predicted NUP98-HOXA9 fusion protein may promote leukaemogenesis through inhibition of HOXA9-mediated terminal differentiation and/or aberrant nucleocytoplasmic transport. Borrow J, Shearman AM, Stanton VP Jr, Becher R, Collins T, Williams AJ, Dube I, Katz F, Kwong YL, Morris C, Ohyashiki K, Toyama K, Rowley J, Housman DE.

HOXA9 as an important human myeloid leukaemia gene and suggest an important role for nucleoporins in human myeloid leukaemia given that a second nucleoporin  Nakamura T, Largaespada DA, Lee MP, Johnson LA, Ohyashiki K, Toyama K, Chen SJ, Willman CL, Chen IM, Feinberg AP, Jenkins NA, Copeland NG, Shaughnessy JD Jr.

NUP98 portion of the fusion protein was shown to be responsible for transforming a clinically silent pre-leukemic phase observed for Hoxa9 into a chronic, stem cell-derived MPD  Kroon E, Thorsteinsdottir U, Mayotte N, Nakamura T, Sauvageau G.

Altered HOX and WNT7A expression in human lung cancer  Calvo R, West J, Franklin W, Erickson P, Bemis L, Li E, Helfrich B, Bunn P, Roche J, Brambilla E, Rosell R, Gemmill RM, Drabkin HA.

DNA binding complexes containing HoxA9 and TALE homeoproteins display cooperative transcriptional activity and are present in leukemic cells  Schnabel CA, Jacobs Y, Cleary ML.

AbdB-like Hox proteins stabilize DNA binding by the Meis1 homeodomain proteins  Shen WF, Montgomery JC, Rozenfeld S, Moskow JJ, Lawrence HJ, Buchberg AM, Largman C.

Hox and Meis are important genes for myeloid leukemogenesis   Fujino T, Yamazaki Y, Largaespada DA, Jenkins NA, Copeland NG, Hirokawa K, Nakamura T.

NUP98-HOXA9 expression in hemopoietic stem cells induces chronic and acute myeloid leukemias in mice  Kroon E, Thorsteinsdottir U, Mayotte N, Nakamura T, Sauvageau G.

Roles of HOX and MEIS1 genes in induction of acute myeloid leukemia  Thorsteinsdottir U, Kroon E, Jerome L, Blasi F, Sauvageau G.

Co-activation of HOXA9 and MEIS1 is a common event in AML  Lawrence HJ, Rozenfeld S, Cruz C, Matsukuma K, Kwong A, Komuves L, Buchberg AM, Largman C.

Hoxa9 transforms primary bone marrow cells through specific collaboration with Meis1a but not Pbx1b  Kroon E, Krosl J, Thorsteinsdottir U, Baban S, Buchberg AM, Sauvageau G.

Biochemical interactions between HoxA9 and TALE homeoproteins mediate cellular transformation in hematopoietic cells Schnabel CA, Jacobs Y, Cleary ML.

HOXA9 and MEIS1 is a common event in AML  Lawrence HJ, Rozenfeld S, Cruz C, Matsukuma K, Kwong A, Komuves L, Buchberg AM, Largman C.

NUP98 was found fused to the HOXA9, HOXD13, or DDX10 genes in human acute myelogenous leukemia  Nakamura T, Yamazaki Y, Hatano Y, Miura I.

CREB binding protein interacts with nucleoporin-specific FG repeats that activate transcription and mediate NUP98-HOXA9 oncogenicity  Kasper LH, Brindle PK, Schnabel CA, Pritchard CE, Cleary ML, van Deursen JM.

Upregulation of Meis1 and HoxA9 in acute lymphocytic leukemias with the t(4 : 11) abnormality Rozovskaia T, Feinstein E, Mor O, Foa R, Blechman J, Nakamura T, Croce CM, Cimino G, Canaani E.

Hoxa9 evokes a cytokine-selective block in differentiation by a mechanism that does not require Meis gene expression or interaction with Pbx through the PIM.  Calvo KR, Sykes DB, Pasillas M, Kamps MP

Expression of HOX genes, HOX cofactors, and MLL in phenotypically and functionally defined subpopulations of leukemic and normal human hematopoietic cells  Kawagoe H, Humphries RK, Blair A, Sutherland HJ, Hogge DE

Upregulation of Meis1 and HoxA9 in acute lymphocytic leukemias with the t(4 : 11) abnormality Rozovskaia T, Feinstein E, Mor O, Foa R, Blechman J, Nakamura T, Croce CM, Cimino G, Canaani E.

NUP98-HOXA9 expression in hemopoietic stem cells induces chronic and acute myeloid leukemias Kroon E, Thorsteinsdottir U, Mayotte N, Nakamura T, Sauvageau G.

co-activation of HOXA9 and MEIS1 is a common event in AML Lawrence HJ, Rozenfeld S, Cruz C, Matsukuma K, Kwong A, Komuves L, Buchberg AM, Largman C.

NUP98/HOXA9 fusion protein could play a critical role in the progression to blast crisis  Yamamoto K, Nakamura Y, Saito K, Furusawa S.

Chronic myelomonocytic leukemia with t(7;11)(p15;p15) and NUP98/HOXA9 fusion Wong KF, So CC, Kwong YL.